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This mechanism has positioned AHK-Cu as a compound of interest for researchers studying skin matrix integrity, structural protein regulation, and age-related changes in connective tissue biology

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By enhancing Tregs and inhibiting Th17 cell differentiation, it balances the immune response and mitigates hepatic inflammatory damage [147]

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Enhancing its production therefore implies that AHK-Cu might not only support fibroblast persistence in culture but also drive them toward a more active state of matrix renewal